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Neurological examination consists of electromyography and electroneurography. Reflexes of different types and sensory sensitivity are evaluated. tactile, vibrational and temperature, also painful. The gastroenterological form is examined using ultrasound and radiography. The genitourinary form involves the study of casodex, the use of an ultrasound machine and muscle electromyography.
- Treatment of diabetic neuropathy takes place in several stages.
- For more effective treatment, compensation for diabetes mellitus is carried out.
- To buy casodex online this result, insulin or antidiabetic tablets are used.
- Also, to optimize treatment, a specially designed diet and systematic physical activity are used.
During the main course of treatment, neurotropic B vitamins and vitamin E antioxidants are used. Also, physiotherapeutic methods of treatment, for example, will not be superfluous. With diabetic neuropathy of the lower extremities, the most scrupulous foot care is required, this includes wearing comfortable shoes, preferably orthopedic, and performing a medical pedicure, and moisturizing the feet.
The detection of diabetic neuropathy in the early stages is quite a promising prognosis for the development of the disease and the normalization of the patient's life. At the initial stage, the symptoms and sensations in diabetic neuropathy may be different, but they can only be reversible when a stable compensation for diabetes is achieved. It is also worth noting the use of alpha-lipoic acid and B vitamins in a low-carbohydrate diet, they do not harm the body, and the benefits of their use are quite significant. The benefits of alpha lipoic acid and its action is to buy bicalutamide online oxidative stress of cells and activate enzymes that block the accumulation of glucose in nerve cells.
Alpha Lipoic Acid repairs damaged nerves. Benfotiamine is also often used, which prevents the toxic effect of glucose on nerve endings.
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Complications of diabetic neuropathy that are at the forefront are painless myocardial infarctions and abnormal heart rhythms. Prevention of diabetic neuropathy requires constant monitoring of blood sugar levels. It also requires timely adjustment of treatment and regular visits to the doctor. Diabetic neuropathy, treatment and symptoms.
In clinical diabetology, damage to the central nervous system is reduced mainly to the diagnosis of disorders of the blood supply to the brain, which is actually a manifestation of macroangiopathy and is discussed in the appropriate section. Violation of the functioning of higher nervous activity, which is associated with metabolic disorders in diabetes, is discussed only in connection with the development of hypoglycemia and diabetic coma.
Neuropathy is one of the most common chronic complications of diabetes mellitus, which can potentially affect any part of bicalutamide pills nervous system, and it manifests itself both in isolation and in combination with other chronic complications of diabetes.
The most typical combination of neuropathic loss of sensation in the foot with an infectious lesion and/or obliterating atherosclerosis of the lower extremities leads to the fact that diabetes mellitus is the most common cause of non-traumatic amputation of the legs in Russia. The frequency of diabetic neuropathy increases with the duration of diabetes mellitus and the degree of hyperglycemia in both type 1 and type 2 diabetes.
The mechanisms that cause neuropathy in diabetes mellitus are realized through hyperglycemia and other metabolic disorders that accompany insulin deficiency, which lead to the activation of the polyol pathway of glucose metabolism, protein glycation, and reduce the intracellular potential for the elimination of peroxidation products. In addition to hyperglycemia and activation of the polyol pathway, oxidative stress, ischemia/hypoxia, disorders of nerve growth factors, and immunological disorders are important in the development of neuropathy.
Histologically, neuropathy is characterized by loss of both large and small myelinated nerve fibers, accompanied by varying degrees of casodex and segmental demyelination, connective tissue proliferation, and thickening and reduplication of the basement membrane of nerve capillaries and their obliteration.
Risk factors for neuropathy include age, duration of diabetes, glycemic control, cholesterol/triglyceride levels, hypertension, microvascular disease, and smoking. In addition, the clinic of diabetic neuropathy does not differ from neuropathy of another etiology (alcoholic, toxic, inflammatory), so its diagnosis is established by exclusion.